Max in WT synaptoneurosomes, suggesting that Src signaling could be downregulated in KI synapses. On the other hand, our ability to rescue SERT functionality in KI midbrain synaptoneurosomes through the inhibition of FAK signifies elevated FAK signaling downstream from the Pro32Pro33 mutant, as verified by enhanced pFAK localization in 5-HT https://gastona097doz8.levitra-wiki.com/user